Varicella-zoster virus: Pathogenesis, immunity, and clinical management in hematopoietic cell transplant recipients BIOLOGY OF BLOOD AND MARROW TRANSPLANTATION Arvin, A. M. 2000; 6 (3): 219-230

Abstract

New information about the mechanisms of varicella-zoster virus (VZV) pathogenesis and the host response to the virus has improved our understanding of the threat that VZV reactivation may pose after hematopoietic cell transplantation (HCT). Antiviral therapy compensates for some of the deficiencies in VZV immunity in HCT recipients, and inactivated varicella vaccine may be useful for the early reconstitution of adaptive immunity to VZV after HCT.

View details for Web of Science ID 000090022300002

View details for PubMedID 10871147