Shirit Einav

Infectious disease doctor

Assistant Professor of Medicine (Infectious Diseases) and of Microbiology and Immunology

Infectious Disease Clinic

  • 900 Blake Wilbur Drive
  • Palo Alto, CA 94304
  • Phone: 650-736-5200
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Professional Education

Residency: Beth Israel Deaconess Med Center/Harvard (2002) MA

Fellowship: Stanford University - Infectious Diseases (2009) CA

Board Certification: Infectious Disease, American Board of Internal Medicine (2006)

Internship: Beth Israel Deaconess Med Center/Harvard (2000) MA

Medical Education: Sackler School of Medicine (1999) Israel

BA, Sackler School of Medicine, Israel, Medical sciences (1994)

MD, Sackler School of Medicine, Israel, Medicine (1999)

Honors & Awards

Research Scholar Grant, American Cancer Society (2014)

Clinical Scientist Development Award, Doris Duke Charitable Foundation (2013)

Stanford Bio-X Interdisciplinary Initiative Program Award, Stanford, Bio-X (2012)

IDSA 2012 IDWeek Investigator Award, Infectious Diseases Society of America (IDSA) (2012)

IDSA 2009 Program Committee Choice Award, Infectious Diseases Society of America (IDSA) (July 2009)

DDC Pilot/Feasibility Award, Stanford Digestive Disease Center (DDC) (March 2009)

ITI Young Investigator Innovation Award, Stanford Institute for Immunity, Transplantation, and Infection (ITI) (Dec 2008)

Mentored Clinical Scientist Development Award (KO8), NIH/NIAID (Sept 2008 - July 2013)

American Liver Foundation Postdoctoral Fellowship Award, American Liver Foundation (ALF) (July 2006)

Dean’s Fellowship Award, Stanford University School of Medicine, Stanford, CA (2004)

Fellow Travel Award, International Symposium on Hepatitis C Virus & Related Viruses (2004)

Excellence in Teaching Housestaff Award, Harvard Medical School, Boston, MA (2002)

Outstanding Thesis Award, Sackler School of Medicine, Tel-Aviv University (1999)

Dean’s Honor List (in four consecutive years), Sackler School of Medicine, Tel-Aviv University (1994-1997)

M.D. graduation magna cum laude, Sackler School of Medicine, Tel-Aviv University (1997)

B.A. graduation magna cum laude, Sackler School of Medicine, Tel-Aviv University (1994)

Administrative Appointments

Assistant Professor (UTL), Department of Medicine, Department of Microbiology and Immunology (2011 - Present)

Instructor of Medicine, Division of Infectious Diseases and Geographic Medicine, Stanford University School of Medicine (2009 - 2011)

Infectious Diseases Fellowship, Division of Infectious Diseases and Geographic Medicine, Stanford University School of Medicine (2004 - 2008)

Postdoctoral Fellowship. Supervisor: Dr. Jeffrey S. Glenn, Division of Gastroenterology and Hepatology, Stanford University School of Medicine (2003 - 2004)

Medical Internship and Residency, Internal Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA (1999 - 2002)

Research Studentship, Supervisor: Dr. Michael C. Carroll., Brigham and Women’s Hospital and the Center for Blood Research, Harvard Medical School, Boston, MA (1999 - 1999)

Rotating internship (part of the M.D. requirements in Israel), Suraski Medical Center, Tel-Aviv, Israel (1997 - 1998)

B-cell receptors expressed by lymphomas of hepatitis C virus (HCV)-infected patients rarely react with the viral proteins
Ng, P. P., Kuo, C.-C., Wang, S., Einav, S., Arcaini, L., & Levy, S. (2014). B-cell receptors expressed by lymphomas of hepatitis C virus (HCV)-infected patients rarely react with the viral proteins. BLOOD, 123(10), 1512-1515.

Identification and Targeting of an Interaction between a Tyrosine Motif within Hepatitis C Virus Core Protein and AP2M1 Essential for Viral Assembly
Neveu, G., Barouch-Bentov, R., Ziv-Av, A., Gerber, D., Jacob, Y., & Einav, S. (2012). Identification and Targeting of an Interaction between a Tyrosine Motif within Hepatitis C Virus Core Protein and AP2M1 Essential for Viral Assembly. PLOS PATHOGENS, 8(8).

The Hepatitis C Virus (HCV) NS4B RNA Binding Inhibitor Clemizole Is Highly Synergistic with HCV Protease Inhibitors
Einav, S., Dvory-Sobol, H., Gehrig, E., & Glenn, J. S. (2010). The Hepatitis C Virus (HCV) NS4B RNA Binding Inhibitor Clemizole Is Highly Synergistic with HCV Protease Inhibitors. JOURNAL OF INFECTIOUS DISEASES, 202(1), 65-74.

A small molecule inhibits HCV replication and alters NS4B's subcellular distribution
Bryson, P. D., Cho, N.-J., Einav, S., Lee, C., Tai, V., & Glenn, J. S. (2010). A small molecule inhibits HCV replication and alters NS4B's subcellular distribution. ANTIVIRAL RESEARCH, 87(1), 1-8.

Six RNA Viruses and Forty-One Hosts: Viral Small RNAs and Modulation of Small RNA Repertoires in Vertebrate and Invertebrate Systems
Parameswaran, P., Sklan, E., Wilkins, C., Burgon, T., Samuel, M. A., & Fire, A. Z. (2010). Six RNA Viruses and Forty-One Hosts: Viral Small RNAs and Modulation of Small RNA Repertoires in Vertebrate and Invertebrate Systems. PLOS PATHOGENS, 6(2).

Discovery of a hepatitis C target and its pharmacological inhibitors by microfluidic affinity analysis
Einav, S., Gerber, D., Bryson, P. D., Sklan, E. H., Elazar, M., & Quake, S. R. (2008). Discovery of a hepatitis C target and its pharmacological inhibitors by microfluidic affinity analysis. NATURE BIOTECHNOLOGY, 26(9), 1019-1027.

The nucleotide binding motif of hepatitis C virus NS4B can mediate cellular transformation and tumor formation without ha-ras co-transfection
Einav, S., Sklan, E. H., Moon, H. M., Gehrig, E., Liu, P., & Glenn, J. S. (2008). The nucleotide binding motif of hepatitis C virus NS4B can mediate cellular transformation and tumor formation without ha-ras co-transfection. HEPATOLOGY, 47(3), 827-835.

TBC1D20 is a Rab1 GTPase-activating protein that mediates hepatitis C virus replication
Sklan, E. H., Serrano, R. L., Einav, S., Pfeffer, S. R., Lambright, D. G., & Glenn, J. S. (2007). TBC1D20 is a Rab1 GTPase-activating protein that mediates hepatitis C virus replication. JOURNAL OF BIOLOGICAL CHEMISTRY, 282(50), 36354-36361.

Mechanisms of resistance to antiviral agents.
Shafer RW, Einav S, & Chou S. (2007). Mechanisms of resistance to antiviral agents. In Manual of clinical microbiology, 9th edition, Murray PR ed, Baron EJ ed, Jorgensen JH ed, Pfaller MA ed, Tenover FC ed, and Yolken RH ed. American Society of Microbiology..

A nucleotide binding motif in hepatitis C virus (HCV) NS4B mediates HCV RNA replication
Einav, S., Elazar, M., Danieli, T., & Glenn, J. S. (2004). A nucleotide binding motif in hepatitis C virus (HCV) NS4B mediates HCV RNA replication. JOURNAL OF VIROLOGY, 78(20), 11288-11295.

Prenylation inhibitors: a novel class of antiviral agents
Einav, S., & Glenn, J. S. (2003). Prenylation inhibitors: a novel class of antiviral agents. JOURNAL OF ANTIMICROBIAL CHEMOTHERAPY, 52(6), 883-886.

Immunopathogenesis of hepatitis C virus in the immunosuppressed host.
Einav, S., & Koziel, M. J. (2002). Immunopathogenesis of hepatitis C virus in the immunosuppressed host. Transplant infectious disease , 4(2), 85-92.

Complement C4 is protective for lupus disease independent of C3
Einav, S., Pozdnyakova, O. O., Ma, M. H., & Carroll, M. C. (2002). Complement C4 is protective for lupus disease independent of C3. JOURNAL OF IMMUNOLOGY, 168(3), 1036-1041.