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Abstract
Acquired resistance to imatinib is a significant problem for the clinical management of gastrointestinal stromal tumor (GIST) patients, and second-line small molecules have shown limited efficacy in this setting. We have recently demonstrated that a monoclonal antibody targeting KIT could potentially bypass imatinib resistance in preclinical models of GIST.
View details for DOI 10.4161/onci.24452
View details for Web of Science ID 000322060900007
View details for PubMedCentralID PMC3716740