Professional Summary
Education & Certifications
- Board Certification: American Board of Internal Medicine, Internal Medicine (2019)
- Visiting scholar, NIH, Undiagnosed Diseases Program (2019)
- Chief Resident, Stanford University Internal Medicine Residency (2019)
- Residency: Stanford University Internal Medicine Residency (2018) CA
- Medical Education: University of Washington School of Medicine (2015) WA
- Ph.D., University of Washington, Molecular and Cellular Biology (2011)
Honors & Awards
- Alpha Omega Alpha (Medical Honor Society), Stanford University chapter (2019)
- Harold M. Weintraub Graduate Student Award (National), Fred Hutchinson Cancer Research Center (2012)
- Julian Wolfsohn Award (for clinical excellence, leadership, teaching, kindness), Stanford Internal Medicine Residency (2016)
- Phi Beta Kappa (Honor Society), Rice University chapter (2006)
Administrative Appointments
- Chief Resident, Stanford Internal Medicine Residency Program (2018 - 2019)
- Co-Director, Stanford Post-Acute COVID-19 Syndrome Clinic (2021 - Present)
- Director, Stanford Consultative Medicine Clinic (2019 - Present)
Publications
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Structural and functional characterization of the first intracellular loop of human thromboxane A(2) receptor
Geng, L., Wu, J. X., So, S. P., Huang, G. X., & Ruan, K. H. (2004). Structural and functional characterization of the first intracellular loop of human thromboxane A(2) receptor. ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS, 423(2), 253–65. -
Variability in the Androgen Response of Prostate Epithelium to 5 alpha-Reductase Inhibition: Implications for Prostate Cancer Chemoprevention
Mostaghel, E. A., Geng, L., Holcomb, I., Coleman, I. M., Lucas, J., True, L. D., & Nelson, P. S. (2010). Variability in the Androgen Response of Prostate Epithelium to 5 alpha-Reductase Inhibition: Implications for Prostate Cancer Chemoprevention. CANCER RESEARCH, 70(4), 1286–95. -
Genomics in medicine: a novel elective rotation for internal medicine residents.
Geng, L. N., Kohler, J. N., Levonian, P., Bernstein, J. A., Ford, J. M., Ahuja, N., … Wheeler, M. (2019). Genomics in medicine: a novel elective rotation for internal medicine residents. Postgraduate Medical Journal. -
Phases of the Diagnostic Journey: A Framework
Geng, L. N., Sum-Ping, O., & Geng, Y.-J. (2019). Phases of the Diagnostic Journey: A Framework. International Archives of Internal Medicine. -
COMMD1 is linked to the WASH complex and regulates endosomal trafficking of the copper transporter ATP7A.
Phillips-Krawczak, C. A., Singla, A., Starokadomskyy, P., Deng, Z., Osborne, D. G., Li, H., … Burstein, E. (2015). COMMD1 is linked to the WASH complex and regulates endosomal trafficking of the copper transporter ATP7A. Molecular Biology of the Cell, 26(1), 91–103. -
DUX4 binding to retroelements creates promoters that are active in FSHD muscle and testis.
Young, J. M., Whiddon, J. L., Yao, Z., Kasinathan, B., Snider, L., Geng, L. N., … Tapscott, S. J. (2013). DUX4 binding to retroelements creates promoters that are active in FSHD muscle and testis. PLoS Genetics, 9(11), e1003947. -
Behcet's disease with major vascular involvement.
Geng, L. N., Conway, D., Barnhart, S., & Nowatzky, J. (2013). Behcet's disease with major vascular involvement. BMJ Case Reports, 2013. -
Generation of isogenic D4Z4 contracted and noncontracted immortal muscle cell clones from a mosaic patient: a cellular model for FSHD.
Krom, Y. D., Dumonceaux, J., Mamchaoui, K., den Hamer, B., Mariot, V., Negroni, E., … van der Maarel, S. M. (2012). Generation of isogenic D4Z4 contracted and noncontracted immortal muscle cell clones from a mosaic patient: a cellular model for FSHD. The American Journal of Pathology, 181(4), 1387–401. -
DUX4 activates germline genes, retroelements, and immune mediators: implications for facioscapulohumeral dystrophy.
Geng, L. N., Yao, Z., Snider, L., Fong, A. P., Cech, J. N., Young, J. M., … Tapscott, S. J. (2012). DUX4 activates germline genes, retroelements, and immune mediators: implications for facioscapulohumeral dystrophy. Developmental Cell, 22(1), 38–51. -
Immunodetection of human double homeobox 4.
Geng, L. N., Tyler, A. E., & Tapscott, S. J. (2011). Immunodetection of human double homeobox 4. Hybridoma (2005), 30(2), 125–30. -
Facioscapulohumeral dystrophy: incomplete suppression of a retrotransposed gene.
Snider, L., Geng, L. N., Lemmers, R. J., Kyba, M., Ware, C. B., Nelson, A. M., … Miller, D. G. (2010). Facioscapulohumeral dystrophy: incomplete suppression of a retrotransposed gene. PLoS Genetics, 6(10), e1001181. -
DNA methylation of developmental genes in pediatric medulloblastomas identified by denaturation analysis of methylation differences.
Diede, S. J., Guenthoer, J., Geng, L. N., Mahoney, S. E., Marotta, M., Olson, J. M., … Tapscott, S. J. (2010). DNA methylation of developmental genes in pediatric medulloblastomas identified by denaturation analysis of methylation differences. Proceedings of the National Academy of Sciences of the United States of America, 107(1), 234–9. -
RNA transcripts, miRNA-sized fragments and proteins produced from D4Z4 units: new candidates for the pathophysiology of facioscapulohumeral dystrophy.
Snider, L., Asawachaicharn, A., Tyler, A. E., Geng, L. N., Petek, L. M., Maves, L., … Tapscott, S. J. (2009). RNA transcripts, miRNA-sized fragments and proteins produced from D4Z4 units: new candidates for the pathophysiology of facioscapulohumeral dystrophy. Human Molecular Genetics, 18(13), 2414–30. -
Loss of Paneth Cell Autophagy Causes Acute Susceptibility to Toxoplasma gondii-Mediated Inflammation.
Burger, E., Araujo, A., López-Yglesias, A., Rajala, M. W., Geng, L., Levine, B., … Yarovinsky, F. (2018). Loss of Paneth Cell Autophagy Causes Acute Susceptibility to Toxoplasma gondii-Mediated Inflammation. Cell Host & Microbe, 23(2), 177–190.e4. -
Deregulation of the protocadherin gene FAT1 alters muscle shapes: implications for the pathogenesis of facioscapulohumeral dystrophy.
Caruso, N., Herberth, B., Bartoli, M., Puppo, F., Dumonceaux, J., Zimmermann, A., … Helmbacher, F. (2013). Deregulation of the protocadherin gene FAT1 alters muscle shapes: implications for the pathogenesis of facioscapulohumeral dystrophy. PLoS Genetics, 9(6), e1003550. -
Type 1 diabetes promotes disruption of advanced atherosclerotic lesions in LDL receptor-deficient mice.
Johansson, F., Kramer, F., Barnhart, S., Kanter, J. E., Vaisar, T., Merrill, R. D., … Bornfeldt, K. E. (2008). Type 1 diabetes promotes disruption of advanced atherosclerotic lesions in LDL receptor-deficient mice. Proceedings of the National Academy of Sciences of the United States of America, 105(6), 2082–7. -
Structure and enzymatic properties of a chimeric bacteriophage RB69 DNA polymerase and single-stranded DNA binding protein with increased processivity.
Sun, S., Geng, L., & Shamoo, Y. (2006). Structure and enzymatic properties of a chimeric bacteriophage RB69 DNA polymerase and single-stranded DNA binding protein with increased processivity. Proteins, 65(1), 231–8. -
Consultative Medicine - An Emerging Specialty for Patients with Perplexing Conditions.
Geng, L. N., Verghese, A., & Tilburt, J. C. (1800). Consultative Medicine - An Emerging Specialty for Patients with Perplexing Conditions. The New England Journal of Medicine, 385(26), 2478–2484. -
Training Internal Medicine Residents in Difficult Diagnosis: A Novel Diagnostic Second Opinion Clinic Experience.
Testa, S., Joshi, M., Lotfi, J., Lin, B., Artandi, M., Chiang, K. F., … Geng, L. N. (2022). Training Internal Medicine Residents in Difficult Diagnosis: A Novel Diagnostic Second Opinion Clinic Experience. Journal of Medical Education and Curricular Development, 9, 23821205221091036. -
Diagnostic journeys: characterization of patients and diagnostic outcomes from an academic second opinion clinic.
Chao, S., Lotfi, J., Lin, B., Shaw, J., Jhandi, S., Mahoney, M., … Geng, L. N. (2022). Diagnostic journeys: characterization of patients and diagnostic outcomes from an academic second opinion clinic. Diagnosis (Berlin, Germany). -
Anti-nucleocapsid antibody levels and pulmonary comorbid conditions are linked to post-COVID-19 syndrome.
Jia, X., Cao, S., Lee, A. S., Manohar, M., Sindher, S. B., Ahuja, N., … Nadeau, K. C. (2022). Anti-nucleocapsid antibody levels and pulmonary comorbid conditions are linked to post-COVID-19 syndrome. JCI Insight, 7(13). -
Characterization of autonomic symptom burden in long COVID: A global survey of 2,314 adults.
Larsen, N. W., Stiles, L. E., Shaik, R., Schneider, L., Muppidi, S., Tsui, C. T., … Miglis, M. G. (2022). Characterization of autonomic symptom burden in long COVID: A global survey of 2,314 adults. Frontiers in Neurology, 13, 1012668. -
The Use of Nirmatrelvir-ritonavir in a Case of Breakthrough Long COVID
Geng, L. N., Bonilla, H. F., Shafer, R. W., Miglis, M. G., & Yang, P. C. (2023). The Use of Nirmatrelvir-ritonavir in a Case of Breakthrough Long COVID. Exploratory Research and Hypothesis in Medicine. -
Myalgic Encephalomyelitis/Chronic Fatigue Syndrome is common in post-acute sequelae of SARS-CoV-2 infection (PASC): Results from a post-COVID-19 multidisciplinary clinic.
Bonilla, H., Quach, T. C., Tiwari, A., Bonilla, A. E., Miglis, M., Yang, P. C., … Geng, L. C. (2023). Myalgic Encephalomyelitis/Chronic Fatigue Syndrome is common in post-acute sequelae of SARS-CoV-2 infection (PASC): Results from a post-COVID-19 multidisciplinary clinic. Frontiers in Neurology, 14, 1090747. -
Development of a Definition of Postacute Sequelae of SARS-CoV-2 Infection.
Thaweethai, T., Jolley, S. E., Karlson, E. W., Levitan, E. B., Levy, B., McComsey, G. A., … Zisis, S. (2023). Development of a Definition of Postacute Sequelae of SARS-CoV-2 Infection. JAMA. -
SARS-CoV-2 reservoir in post-acute sequelae of COVID-19 (PASC).
Proal, A. D., VanElzakker, M. B., Aleman, S., Bach, K., Boribong, B. P., Buggert, M., … Wherry, E. J. (2023). SARS-CoV-2 reservoir in post-acute sequelae of COVID-19 (PASC). Nature Immunology. -
Low-dose naltrexone use for the management of post-acute sequelae of COVID-19.
Bonilla, H., Tian, L., Marconi, V. C., Shafer, R., McComsey, G. A., Miglis, M., … Geng, L. N. (2023). Low-dose naltrexone use for the management of post-acute sequelae of COVID-19. International Immunopharmacology, 124(Pt B), 110966. -
Features of acute COVID-19 associated with post-acute sequelae of SARS-CoV-2 phenotypes: results from the IMPACC study.
Ozonoff, A., Jayavelu, N. D., Liu, S., Melamed, E., Milliren, C. E., Qi, J., … Rouphael, N. (2024). Features of acute COVID-19 associated with post-acute sequelae of SARS-CoV-2 phenotypes: results from the IMPACC study. Nature Communications, 15(1), 216. -
IgM N-glycosylation correlates with COVID-19 severity and rate of complement deposition.
Haslund-Gourley, B. S., Woloszczuk, K., Hou, J., Connors, J., Cusimano, G., Bell, M., … Comunale, M. A. (2024). IgM N-glycosylation correlates with COVID-19 severity and rate of complement deposition. Nature Communications, 15(1), 404. -
New Alcohol Sensitivity in Patients With Post-acute Sequelae of SARS-CoV-2 (PASC): A Case Series.
Eastin, E. F., Tiwari, A., Quach, T. C., Bonilla, H. F., Miglis, M. G., Yang, P. C., & Geng, L. N. (2023). New Alcohol Sensitivity in Patients With Post-acute Sequelae of SARS-CoV-2 (PASC): A Case Series. Cureus, 15(12), e51286.
Clinical Trials
Clinical trials are research studies that evaluate a new medical approach, device, drug, or other treatment. As a Stanford Health Care patient, you may have access to the latest, advanced clinical trials.
Open trials refer to studies currently accepting participants. Closed trials are not currently enrolling, but may open in the future.
Practice Locations
(301) Post-Acute COVID-19 Syndrome (PACS) Clinic Atherton, CA
Atherton, CA(301) Post-Acute COVID-19 Syndrome (PACS) Clinic
3351 El Camino Real, Suite 225
Atherton , CA 94027
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Stanford Health Care, Stanford Health Care Tri-Valley, and Stanford Medicine Partners are each independent nonprofit organizations that are affiliated with but separate from each other and from Stanford University. The physicians who provide care at facilities operated by Stanford Health Care, Stanford Health Care Tri-Valley, and Stanford Medicine Partners are faculty, foundation, or community physicians who are not employees, representatives, or agents of Stanford Health Care, Stanford Health Care Tri- Valley, or Stanford Medicine Partners. Stanford Health Care, Stanford Health Care Tri-Valley, and Stanford Medicine Partners do not exercise control over the care provided by such faculty, foundation, and community physicians and are not responsible for their actions.
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